(1987): Residual cell-mediated immunity to recall antigens in pediatric AIDS-related disease. (1986): Longitudinal study of 18 children with perinatal LAV/HTLV III infection: attempt at prognostic evaluation. (1985): Defective humoral immunity in pediatric acquired immune deficiency syndrome. titers in the months following the end of the primary cycle.īernstein L.J., Ochs H.D., Wedgwood R.J.et al. titers in those HIV infected children who showed significant decreases of n.a. Interestingly, the recall dose was also able to induce high n.a. Infected and non-infected subjects responded to the same extent with high levels of n.a. Sixteen children also received a booster dose of vaccine one year after the completion of the primary cycle. The poorest response was observed in subjects with full-blown immunodeficiency (CD4 < 1000/mm 3, reduced response to PWM). positive subjects and as antibody levels were found between HIV infected children and those who lost HIV antibodies. No statistically significant differences both as rates of n.a.
All these subjects have been followed for 32 (15–48) months in order to ascertain their infection status.Īfter a course of 2 doses of E-IPV, 88% of children had neutralizing antibody (n.a.) titers > 1:4 to the 3 poliovirus serotypes and 100% to at least 2 polio strains. In order to evaluate the response to immunization of HIV-infected children we studied the humoral response to an enhanced potency inactivated poliovaccine (E-IPV) of 43 children born of HIV seropositive mothers.
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